| Bioactivity | Ac2-26, an active N-terminal peptide of annexin A1 (AnxA1), attenuates ischemia-reperfusion-induced acute lung injury. Ac2-26 also decreases AnxA1 protein expression, inhibits the activation of NF-κB and MAPK pathways in the injured lung tissue[1]. | ||||||
| Name | Ac2-26 | ||||||
| CAS | 151988-33-9 | ||||||
| Sequence | Ac-Ala-Met-Val-Ser-Glu-Phe-Leu-Lys-Gln-Ala-Trp-Phe-Ile-Glu-Asn-Glu-Glu-Gln-Glu-Tyr-Val-Gln-Thr-Val-Lys | ||||||
| Shortening | Ac-AMVSEFLKQAWFIENEEQEYVQTVK | ||||||
| Formula | C141H210N32O44S | ||||||
| Molar Mass | 3089.43 | ||||||
| Appearance | Solid | ||||||
| Transport | Room temperature in continental US; may vary elsewhere. | ||||||
| Storage | Sealed storage, away from moisture and light
*In solvent : -80°C, 6 months; -20°C, 1 month (sealed storage, away from moisture and light) |
||||||
| Reference | [1]. Liao WI, et al. Ac2-26, an Annexin A1 Peptide, Attenuates Ischemia-Reperfusion-Induced Acute Lung Injury. Int J Mol Sci. 2017 Aug 15;18(8). pii: E1771. |
Ac2-26
CAS: 151988-33-9 F: C141H210N32O44S W: 3089.43
Ac2-26, an active N-terminal peptide of annexin A1 (AnxA1), attenuates ischemia-reperfusion-induced acute lung injury. A
Data collection：peptidedb@qq.com
This product is for research use only, not for human use. We do not sell to patients.