| Bioactivity | β Amyloid(28-35) human is a β-amyloid peptide (Abeta), a lipid-induced amyloid core fragment. β Amyloid oligomers are neurotoxic, and β Amyloid(28-35) human can interact with neuronal membranes, regulate secondary structure and neurotoxicity, and cause Alzheimer's disease. β Amyloid(28-35) human has anisotropic effects on the acidic phospholipid DPH, resulting in enhanced internal fluidity of lipid membrane bilayers[1]. |
| Name | β Amyloid(28-35) human |
| CAS | 182945-56-8 |
| Sequence | Lys-Gly-Ala-Ile-Ile-Gly-Leu-Met |
| Shortening | KGAIIGLM |
| Formula | C36H67N9O9S |
| Molar Mass | 802.04 |
| Transport | Room temperature in continental US; may vary elsewhere. |
| Storage | Please store the product under the recommended conditions in the Certificate of Analysis. |
| Reference | [1]. Nagarajan S, et al. Lipid-induced conformational transition of the amyloid core fragment Abeta(28-35) and its A30G and A30I mutants. FEBS J. 2008 May;275(10):2415-27. |
β Amyloid(28-35) human
CAS: 182945-56-8 F: C36H67N9O9S W: 802.04
β Amyloid(28-35) human is a β-amyloid peptide (Abeta), a lipid-induced amyloid core fragment. β Amyloid oligomers are
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