| Bioactivity | Paclitaxel is a naturally occurring antineoplastic agent and stabilizes tubulin polymerization. Paclitaxel can cause both mitotic arrest and apoptotic cell death. Paclitaxel also induces autophagy[1][2]. |
| Invitro | Paclitaxel (20 nM; 48 hours) induces programmed cell death and exists a block at the G2/M phase of the cell cycle[1].Paclitaxel (20 nM; 48 hours) induces a consistent increase in the level of p53[1]. Apoptosis Analysis[1] Cell Line: |
| Name | Paclitaxel |
| CAS | 33069-62-4 |
| Formula | C47H51NO14 |
| Molar Mass | 853.91 |
| Transport | Room temperature in continental US; may vary elsewhere. |
| Storage | 4°C, protect from light *In solvent : -80°C, 6 months; -20°C, 1 month (protect from light) |
Paclitaxel
CAS: 33069-62-4 F: C47H51NO14 W: 853.91
Paclitaxel is a naturally occurring antineoplastic agent and stabilizes tubulin polymerization. Paclitaxel can cause bot
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