| Bioactivity | Dipentyl phthalate is an endocrine-disrupting phthalate plasticizer. Dipentyl phthalate increases AMPK phosphorylation and decreases AKT1 phosphorylation and SIRT1 levels. Dipentyl phthalate reduces adrenocorticotropic hormone levels. Dipentyl phthalate is a testicular toxicant[1]. | ||||||||||||
| Name | Dipentyl phthalate | ||||||||||||
| CAS | 131-18-0 | ||||||||||||
| Formula | C18H26O4 | ||||||||||||
| Molar Mass | 306.40 | ||||||||||||
| Appearance | Liquid | ||||||||||||
| Transport | Room temperature in continental US; may vary elsewhere. | ||||||||||||
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| Reference | [1]. Haiqiong Chen,et al. Exposure to dipentyl phthalate in utero disrupts the adrenalcortex function of adult male rats by inhibiting SIRT1/PGC-1α and inducing AMPKphosphorylation. Environ Toxicol. 2023 May;38(5):997-1010. [2]. Bethany R Hannas,et al. Dipentyl phthalate dosing during sexual differentiation disruptsfetal testis function and postnatal development of the male Sprague-Dawley ratwith greater relative potency than other phthalates. Toxicol Sci. 2011Mar;120(1):184-93. |
Dipentyl phthalate
CAS: 131-18-0 F: C18H26O4 W: 306.40
Dipentyl phthalate is an endocrine-disrupting phthalate plasticizer. Dipentyl phthalate increases AMPK phosphorylation a
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