PeptideDB

Colivelin

CAS No.: 867021-83-8

Colivelin is a hybrid peptide which mediates neuroprotection against Alzheimer’s disease (AD)-relevant insults. It is c
Sales Email:peptidedb@qq.com

This product is for research use only, not for human use. We do not sell to patients.

CAS 867021-83-8
Sequence H-Ser-Ala-Leu-Leu-Arg-Ser-Ile-Pro-Ala-Pro-Ala-Gly-Ala-Ser-Arg-Leu-Leu-Leu-Leu-Thr-Gly-Glu-Ile-Asp-Leu-Pro-OH
Sequence Single SALLRSIPAPAGASRLLLLTGEIDLP
Molecular Formula C119H206N32O35
Molecular Weight 2645.14
Technology Synthetic
Storage -20°C, avoid light, cool and dry place
Application Alzheimer’s Disease
Description Colivelin is a hybrid peptide which mediates neuroprotection against Alzheimer’s disease (AD)-relevant insults. It is composed of activity-dependent neurotrophic factor (ADNF) C-terminally fused to AGA(C8R)HNG17, a potent humanin derivative. Colivelin completely suppressed death induced by overexpressing familial AD-causative genes and amyloid β-protein (1-43) at a concentration of 100 fM. Colivelin is a brain penetrant neuroprotective peptide and a potent activator of STAT3, suppresses neuronal death by activating STAT3 in vitro. Colivelin exhibits long-term beneficial effects against neurotoxicity, Aβ deposition, neuronal apoptosis, and synaptic plasticity deficits in neurodegenerative disease. Colivelin has the potential for the treatment of alzheimer’s disease and ischemic brain injury.
References 1.  Chiba et al (2005) Development of a femtomolar-acting humanin derivative named colivelin by attaching activity-dependent neurotrophic factor to its N terminus: characterization of colivelin-mediated neuroprotection against Alzheimer’s disease-relevant insults in vitro. Tomohiro Chiba et al., Neurosci. 25 10252 PMID: 16267233 2.  A novel peptide, colivelin, prevents alcohol-induced apoptosis in fetal brain of C57BL/6 mice: signaling pathway investigations. Sari et al (2009), Neuroscience 164 1653 PMID: 19782727 3.  Nasal colivelin treatment ameliorates memory impairment related to Alzheimer’s disease.  Yamada et al (2008). Neuropsychopharmacology 33 2020 PMID: 17928813