PeptideDB

Cetrorelix

CAS No.: 120287-85-6

Cetrorelix is a potent gonadotrophin-releasing hormone (GnRH) antagonist. Cetrorelix inhibits the endogenous luteinizing
Sales Email:peptidedb@qq.com

This product is for research use only, not for human use. We do not sell to patients.

CAS 120287-85-6
Sequence Ac-D-2-Nal-4-chloro-D-Phe-β-(3-pyridyl)-D-Ala-Ser-Lys(nicotinoyl)-D-Lys(nicotinoyl)-Leu-Lys(isopropyl)-Pro-D-Ala-NH2
Molecular Formula C70H92ClN17O14
Molecular Weight 1431.06
Technology Synthetic
Storage -20°C, avoid light, cool and dry place
Application Cancer Research|Pituitary & Hypothalamic Hormones|Veterinary Medicine
Description Cetrorelix is a potent gonadotrophin-releasing hormone (GnRH) antagonist. Cetrorelix inhibits the endogenous luteinizing hormone surge during ovarian stimulation. Cetrorelix reduces cyclophosphamide induced ovarian follicular destruction in mice.
References 1.  Mutational analysis of a human immunodeficiency virus type 1 Tat protein transduction domain which is required for delivery of an exogenous protein into mammalian cells. J.Park et al., J. Gen. Virol., 83, 1173 (2002) 2.  GnRH antagonist in in vitro fertilization: where we are now. D.B.Shapiro and D.Mitchell-Leef, Minerva Ginecol., 55, 373 (2003) 3.  The LHRH antagonist cetrorelix: a review. T.Reissmann et al., Hum. Reprod. Update, 6, 322 (2000) 4.  Antiproliferative effects of the GnRH antagonist cetrorelix and of GnRH-II on human endometrial and ovarian cancer cells are not mediated through the GnRH type I receptor. C.Gründker et al., Eur. J. Endocrinol., 151, 141 (2004) 5.  GnRH antagonists in the treatment of gynecological and breast cancers. G.Emons et al., Endocr. Relat. Cancer, 10, 291 (2003) 6.  Suppression of the endogenous luteinizing hormone surge by the gonadotrophin-releasing hormone antagonist Cetrorelix during ovarian stimulation. Diedrich K, et al. Hum Reprod. 1994 May;9(5):788-91. 7.  The GnRH antagonist cetrorelix reduces cyclophosphamide-induced ovarian follicular destruction in mice. Meirow D, et al. Hum Reprod. 2004 Jun;19(6):1294-9.