| Description | β-Amyloid (31-35) (β-Amyloid 31-35) is the shortest sequence of native Amyloid-β peptide. |
| Synonyms | 异亮氨酰-异亮氨酰-甘氨酰-亮氨酰-蛋氨酸, β-Amyloid 31-35(TFA), β-Amyloid 31-35 |
| molecular weight | 545.74 |
| Molecular formula | C25H47N5O6S |
| CAS | 149385-65-9 |
| Storage | keep away from moisture | Powder: -20°C for 3 years | In solvent: -80°C for 1 year |
| Solubility | DMSO: 10 mM |
| References | 1. Misiti F , Clementi M E , Tringali G , et al. fragment 31–35 of β-amyloid peptide induces neurodegeneration in rat cerebellar granule cells via bax gene expression and caspase-3 activation: a crucial role for the redox state of methionine-35 residue[J]. Neurochemistry International, 2006, 49(5):525-532. 2. Milton N G N . The amyloid-β peptide binds to cyclin B1 and increases human cyclin-dependent kinase-1 activity[J]. Neuroscience Letters, 2002, 322(2):0-133. 3. Milton NG, et al. The amyloid-beta peptide binds to cyclin B1 and increases human cyclin-dependent kinase-1 activity. Neurosci Lett. 2002 Apr 5;322(2):131-3. |