| Bioactivity | KT-362 is an intracellular calcium antagonist with antiarrhythmic and vasodilatory effects. KT-362 shows an antagonistic effect against norepinephrine (NE) induced vasoconstriction response, achieved by reducing inositol phospholipid hydrolysis, thereby reducing intracellular calcium mobilization. KT-362 can be used to study the contraction and relaxation mechanisms of vascular smooth muscle, especially in exploring the role of intracellular calcium mobilization and inositol phospholipid hydrolysis in vascular contraction[1]. |
| CAS | 93392-97-3 |
| Formula | C22H28N2O3S |
| Molar Mass | 400.53 |
| Transport | Room temperature in continental US; may vary elsewhere. |
| Storage | Please store the product under the recommended conditions in the Certificate of Analysis. |
| Reference | [1]. Kodama I, et al. Effects of KT-362, a new antiarrhythmic agent with vasodilating action on intracellular calcium mobilization of atrial muscle[J]. Journal of Pharmacology and Experimental Therapeutics, 1991, 258(1): 332-338. |
KT-362 free acid
CAS: 93392-97-3 F: C22H28N2O3S W: 400.53
KT-362 is an intracellular calcium antagonist with antiarrhythmic and vasodilatory effects. KT-362 shows an antagonistic
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