| Bioactivity | Dehydroascorbic acid, a blood-brain barrier transportable form of vitamin C, mediates potent cerebroprotection in experimental stroke. | |||||||||
| Invitro | Neuronal injury in ischemic stroke is partly mediated by cytotoxic reactive oxygen species. Although the antioxidant ascorbic acid (AA) or vitamin C does not penetrate the blood-brain barrier (BBB), its oxidized form, dehydroascorbic acid (DHA), enters the brain by means of facilitative transport[1]. | |||||||||
| Name | Dehydroascorbic acid | |||||||||
| CAS | 490-83-5 | |||||||||
| Formula | C6H6O6 | |||||||||
| Molar Mass | 174.11 | |||||||||
| Transport | Room temperature in continental US; may vary elsewhere. | |||||||||
| Storage |
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